By Allan I. Basbaum (auth.), T. H. Stanley, M. A. Ashburn (eds.)
Anesthesiology and soreness Management comprises the edited displays of the thirty ninth Annual Postgraduate path in Anesthesiology, February 1994. The chapters replicate new info thoughts in the basic framework of `pain learn and easy science', and `clinical themes in ache management'. the aim of the textbook is to function a motor vehicle to carry some of the most modern strategies in anesthesiology to those that didn't attend the convention, inside of a short while of the formal presentation. each one bankruptcy is a short yet sharply concentrated glimpse of the present pursuits in anesthesia. This quantity, in addition to prior and destiny volumes, displays the fast and carrying on with evolution of anesthesiology within the past due 20th century.
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Additional info for Anesthesiology and Pain Management
Other studies, in addition, have reported decreased thresholds of dorsal horn neurons to mechanical stimulation following inflammation and injury (28,30). \\\'\'\\] AFTER MK-«11 Figure 2. Changes in the receptive fields of superficial dorsal horn neurons produced by inflammation and drug manipulations. The first row illustrates examples of the small receptive fields found in control animals. The second row shows examples of enlarged receptive fields after inflammation. The third row illustrates the small enlargement of the receptive fields in control animals following the application of dynorphin peptide to the surface of the spinal cord.
How does the expansion of the receptive fields of nociceptive neurons lead to hyperalgesia? One hypothesis (1) is that expanded receptive fields will result in greater overlap of the receptive fields of neurons and, therefore, will lead to a greater number of neurons activated by a stimulus than the number activated by the same stimulus applied in the absence of receptive field expansion. The increase in neuronal activity may ultimately be perceived as more intense pain. It appears that both the observed changes in receptive field size of superficial dorsal horn neurons and the alterations in dynorphin gene expression are closely correlated with the development of behavioral hyperalgesia.
Related studies have also reported facilitation or inhibition of dorsal horn neuronal activity after the administration of kappa opioid receptor agonists (34,35). The two types of responses may represent activation of two functionally different populations of neurons, or alternatively, the two responses may reflect interaction of the agonists at more than one receptor type. Recent data support the argument that kappa opioid receptor agonists have a role in the antinociceptive response to inflammatory pain (36).